This page has links to descriptions of primary research papers written by us since 2007, together with key images which we feel help explain their contents.
Although not "lay summaries", these synopses require less background knowledge of HCV biology than the papers themselves. We hope they're useful. If you want us to explain anything that's unclear (or just to comment on the data), please send us an email.
If you prefer, we now have the list below (up to the end of 2013) as an e-book, which you can download in either Kindle
or EPub (iPad, Android etc.) format (we don't actually have a Kindle, so we haven't checked that file).
Each page below also has a direct PDF download link for every paper which we're allowed to give away (subject to restrictions set by the publisher), you'll find a link on every page. For all our other papers we're happy to supply PDFs by email on request. You'll also find a lot of them HERE.
We also have a page of review articles you can access by clicking HERE.
Click here for a (much longer) list of every paper on which we've been co-authors, our current h-index and other statistics.
- Type I interferon rapidly restricts infectious hepatitis C virus particle genesis Meredith et al. Hepatology, 2014
- Activated Macrophages Promote Hepatitis C Virus Entry in a Tumor Necrosis Factor-Dependent Manner
Fletcher et al. Hepatology, 2014
- Paracrine signals from liver sinusoidal endothelium regulate hepatitis C virus replication
Rowe et al. Hepatology, 2014
Early infection events highlight the limited transmissibility of hepatitis C virus in vitro.
Meredith et al. Hepatology, 2013
- Heterogeneous Claudin-1 Expression in Human Liver.
Harris, et al. Hepatology, 2013.
Hepatoma polarization limits CD81 and hepatitis C virus dynamics.
Harris et al. Cellular Microbiology, 2013
In silico directed mutagenesis identifies the CD81/claudin-1 hepatitis C virus receptor interface.
Davis et al. Cellular Microbiology, 2012
- Hepatitis C virus induces CD81 and claudin-1 endocytosis
Farquhar et al. Journal of Virology, 2012
A dual role for hypoxia inducible factor-1α in the hepatitis C virus lifecycle and hepatoma migration. Wilson et al, Journal of Hepatology, 2012
- Hepatitis C Virus Infects the Endothelial Cells of the Blood–Brain Barrier. Fletcher et al, Gastroenterology, 2012
Neutralizing antibody resistant hepatitis C virus cell-to-cell transmission.
Brimacombe, Grove, et al. J. Virology 2011.
Hepatitis C virus infection of neuroepithelioma cell lines. Fletcher et al, Gastroenterology, 2010
- Claudin association with CD81 defines hepatitis C virus entry. Harris et al, J. Biol. Chem, 2010
- Hepatitis C virus infection reduces hepatocellular polarity in a vascular endothelial growth factor dependent manner. Mee et al, Gastroenterology, 2010
- Hepatoma cell density promotes Claudin-1 and SR-BI expression and hepatitis C virus internalization Schwarz et al, J. Virology 2009
- Polarization restricts hepatitis C virus entry into HepG2 hepatoma cells Mee et al, J. Virology 2009
- Hepatitis C virus association with peripheral blood B lymphocytes potentiates viral infection of liver derived hepatoma cells Stamataki et al, Blood, 2009
- Identification of a residue in hepatitis C virus E2 glycoprotein that determines scavenger receptor BI and CD81 receptor dependency and sensitivity to neutralizing antibodies Grove et al, J. Virology 2008
- Protein Kinase A dependent step(s) in Hepatitis C virus entry and infectivity Farquhar et al, J. Virology 2008
- CD81 and Claudin 1 co-receptor association: a role in hepatitis C virus entry Harris et al, J. Virology 2008
- Effect of cell polarization on hepatitis C virus entry Mee et al, J. Virology 2008
- Hepatitis C virus cell-cell transmission in hepatoma cells in the presence of neutralizing antibodies Timpe et al, Hepatology 2008
- Hepatitis C virus receptor expression in normal and diseased liver tissue Reynolds, Harris et al, Hepatology 2008
- Hepatitis C virus envelope glycoprotein immunization of rodents elicits cross-reactive neutralizing antibodies Stamataki et al, Vaccine 2007
- Scavenger receptor BI and BII expression levels modulate hepatitis C virus infectivity Grove et al, J. Virology 2007