Our research focuses on understanding the mechanism of hepatitis C virus (HCV) entry into the liver and how the virus persists to evade the innate and adaptive immune responses. Development of multi-cellular systems comprising hepatocytes and non-parenchymal liver cells have enabled us to uncover new pathways that regulate viral replication. We are exploring the impact of viral and bacterial co-infection on HCV pathogenesis and the role persistent HCV replication plays in the development of hepatocellular carcinoma.
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